Renal Health

Kidney Stone Removal – What to Expect

Nowadays, the prevalence of kidney stones (also known as renal calculus/calculi) has risen, especially in modern society.

Doctors prefer for patients to pass small enough kidney stones themselves through the act of urination, however the human body often doesn’t take our preferences into consideration. Frequently, patients are unable to pass kidney stones due to the simple fact that they have grown too big. Without surgical intervention, patients will experience excruciating pain as well as possible life threatening complications in the inevitable event of a blockage.

It is extremely uncommon for a stone situated in the kidney to cause pain. This becomes an entirely different story when the stone – much like a ripe apple falling from a tree – detaches itself from the interior of the kidney and embarks on a journey to the bladder which is connected to the kidney via a small tube called the ureter. Unfortunately, if the stone is too large to pass it will most assuredly become lodged somewhere along the way, or become stuck on the wall of the ureter; which can have catastrophic and immediate painful ramifications. When the stone lodges in the ureter, the patient experiences colic, resulting in excruciating pain as well as possible medical complications.

Pain caused by kidney stones has been described as one of the worst pains a human can experience.

Possible outcomes of a renal blockage…

  • Kidney infection
  • Kidney damage
  • Kidney failure
  • Hydronephrosis – When the kidneys are unable to drain urine to the bladder – the urine accumulates in the kidneys. As a result the organ becomes engorged (swollen), which is excruciatingly painful and if left untreated will cause kidney failure and death. The kidney may rupture, relieving the pressure and preserving the organ. Otherwise the raised pressure will gradually destroy the kidney, usually within a couple of months.

The initial treatment is almost always analgesia with narcotic agents and anti-spasmodics, then a scan to assess the position and size of the stone. The urologist will make a decision at that point as to whether the stone will pass with or without surgical intervention.

Obviously, the best solution is when the patient can expel the stone(s) naturally, often with the assistance of medication for nausea and analgesics/muscle relaxants to ease the passage of the stone. Patients will be asked to void urine into a cup-like sieve to catch the stone. This allows the patient to see when the stone comes out and allows for the stone to be collected in order to be sent to a laboratory for analysis. Knowing the composition of the stone assists in preventing the growth of future stones.

If an operative solution is required, the technique used will depend on; stone position, size, hardness and the experience of the urologist.

Operative solutions…

Here are some root word definitions in order to facilitate the reader with understanding the various procedures.

Eg. Nephrolithotomy = Kidney stone removal

  1. “Nephro-“ = Kidney
  2. “-Litho” = Stone
  3. “-Tomy” = Removal
  4. “-Tripsy” = Crushed
  5. “Lapara-“ = Abdomen (Soft part of abdomen between the ribs and hips)
  6. “-Scope” = To see
  7. Extracorporeal = From outside the body

Ureteral Stents

In medicine, the word “stent” can be defined as a temporary splint that is placed within a blood vessel, duct or canal in order to facilitate healing and/or alleviate a blockage.

Rendering of a Double J stent
Simple enlarged rendering of a Double J stent

A ureteral stent is a thin, hollow, flexible tube that surgeons insert into the ureter in order to prevent any blockages forming between the kidney and the bladder. A stent is normally inserted after a kidney stone procedure, it helps maintain adequate drainage of fluid from the kidney to the bladder. The stent also ensures that any stones passing from the kidney to the bladder, don’t become lodged in the ureter. The ureteral stents are commonly called, “double J stents” as each end of the stent is curled in order to ensure they remain in place.

There are various types of stents, all serve the same purpose but what sets them apart is the length of time they can be used for. Double J stents are intended for short term use – ideally a few weeks. Whereas, “indwelling” stents such as, Allium stents; can be left in for up to three years.

Risks associated with ureteral stents

  • Increased risk of ureteral infection


A minimally invasive method of removing stones from a patient’s ureter, especially ones lodged close to the bladder. Commonly performed in the event that ESWL fails. A ureteroscopy is commonly performed under general anaesthesia.

A long, thin, flexible tube known as an ureteroscope (essentially the same as an endoscope) is passed through the urethra and bladder in order to access the ureter(s). A ureteroscope has a light and camera on one end so surgeons are able to visualise the inside of the patient without making a single incision.

There are two types of ureteroscope; semi-rigid and flexible. Semi-rigid scopes are often used for removing stones in the lower ureter, but can be used in the upper ureter if it is dilated enough to allow safe passage of the semi-rigid scope. A flexible scope is safer and less likely to cause trauma above the pelvic brim.

A flexible ureteroscope is used to remove stones in the kidney too small to address with ESWL.

Once the stone has been located, the urologist will discern whether or not the stone is small enough to be removed whole; or if it needs to be fragmented using a laser.

An instrument called a “basket” can be introduced along side the ureteroscope in order to grab and remove any stones or debris from the kidney, ureter, bladder or urethra. The basket resembles the wire end of a whisk, which can be extended and retracted inside the patient safely and with ease. Some patients may also require the insertion of a stent after the procedure; at the discretion of the urologist.

Examples of fully extended baskets

Patients can expect to be discharged on the same day as the procedure, provided there were no complications or unforeseen findings. Typically, regular daily activities can be resumed 2-3 days after the procedure.

Risks associated with ureteroscopy
  • Ureteral stent discomfort
  • Ureteral wall injury
  • Stone migration
  • Infection
  • Bleeding

Extracorporeal Shock Wave Lithotripsy (ESWL)

Extracorporeal shock wave lithotripsy, or ESWL is the least invasive form of kidney/ureteral stone removal; performed under general anaesthesia.  

This treatment modality relies on a machine called a lithotripter to generate high-energy shockwaves that are directed at the kidney/ureteral stone. As a result, the shockwaves reduce the stone(s) to smaller fragments; the goal is to reduce stones to small enough sizes in order for the patient to safely expel them through urination.

For this nonsurgical treatment, the shockwaves are administered to the body via a soft water filled casing. The patient is positioned with the area intended for treatment placed against the casing which pulsates with each shockwave.

Simple diagram of lithotripter

If the surgeon feels that the stone fragments are still too large to safely and comfortably pass, a uretroscopy can easily be performed to remove the fragmented stone. Doctors prefer ESWL as it eliminates the need to make any incisions in the patient. 

Patients can expect to be discharged on the same day as the procedure, provided there were no complications or unforeseen findings. Typically, regular daily activities can be resumed 2-3 days after the procedure

What makes a patient unsuitable for ESWL

Being the least invasive treatment modality for kidney stone removal, doctors would ideally rely on ESWL the most. However, there are certain factors that can make a patient an unsuitable candidate for ESWL.

  • Obesity
  • Pregnancy
  • Bleeding disorders
  • Severe skeletal abnormalities
  • Kidney cancer
Practitioners may be hesitant to suggest ESWL if…

Some factors may not exclude patients as candidates entirely, however practitioners may be cautious when considering ESWL as a treatment option. These factors include…

  • Chronic kidney infection
  • Scar tissue in the ureter
  • Stones consisting of cystine or calcium
  • Stones that need to be removed immediately
  • Having a cardiac pacemaker
Risks associated with ESWL
  • Bleeding around the kidney
  • Urinary tract infection
  • Blockage of urinary tract from stone fragments

Percutaneous Nephrolithotomy (PCNL)

A percutaneous nephrolithotomy (PCNL/PNL) is a form of treatment generally reserved for the larger kidney stones (20mm and bigger) or “staghorn” stones. Practitioners will also opt for this surgery in the event of ESWL/ureteroscopy failing or proving to not be viable. PCNL is performed with the patient under general anaesthesia.

In order to perform a PCNL, an incision is made in the patients flank over the kidney. A guide wire is inserted into the kidney under x-ray guidance through the incision. Graduated sheaths are placed over the guide wire to incrementally enlarge a tract directly to the kidney from the skin. A nephroscope is introduced into the kidney along the tract and the stone can the be visualised.

If the stones happen to be too large to remove whole, a laser can be used to reduce the stone to smaller fragments.

Patients can anticipate a 2-3 day long hospital stay after the procedure. Patient’s may require 1-2 weeks to recover in totality from the procedure.

Risks associated with PCNL
  • Bleeding
  • Renal pelvis perforation (Formation of a hole in the kidney)
  • Hydrothorax (Accumulation of fluid around the lungs)
  • Infection

Laparoscopic Ureterolithotomy

The main purpose of this procedure is to remove larger stones lodged in the ureter. In most cases, this procedure is only performed when less invasive methods fail. Laparoscopic procedures are performed under general anaesthesia.

Small incisions (generally 0.5-1.5cm long) are made in the patient’s abdomen, into which a laparoscope and carbon dioxide gas supplying tube is inserted. The laparoscope is similar to that of a endoscope, however they differ in that the optic tube is not flexible. The reason for filling the abdomen with gas, is to create more space which makes it easier for the surgeon to access and visualise the patient’s internal organs.

Additional small incisions will be made in order to insert the operative instruments. The surgeon will use a cutting and a grasping tool. Using the optic scope, the surgeon locates the affected kidney and ureter; a small incision is made in the ureter in order to remove the ureteral stones. Once all stones have been removed, the ureter is then stented and sutured closed.

Once the operation is completed the gas is released from the abdomen of the patient and all small incisions in the abdomen are sutured closed.

Patients can expect a hospital stay of 2-3 days granted no complications arose, and regular daily activities can be resumed within 2-3 weeks.

Open Surgery

Open surgery for kidney stones is the most invasive form of treatment and is considered to be the last resort if all other options fail. Open surgery is performed under general anaesthesia

Open surgery is only performed when alternative stone removal methods fail or prove to be insufficient. Surgeons may also have to resort to open surgery if the stones are abnormally large, especially in the case of “staghorn” stones. Patients born with a defect in their urinary system may require open surgery too.

To perform this surgery; once the patient is safely asleep, a large enough incision is made in the patient’s abdomen or side in order to expose the affected kidney and ureter. Wound retractors keep the surgical site open whilst the team operates. The surgeon will then make an incision in the organ, allowing access to the stones that need to be removed.

Once all stones have been removed, and any repair work is completed; the surgeon sutures the kidney closed and then does the same for the abdominal/flank incision.

Prior to closing, a catheter may need to be placed in the kidney. By doing so, urine can be drained from the kidney as the organ heals.

Patients can expect a hospital stay of 6-9 days and a recovery period of 4-6 weeks.

Risks associated with open surgery
  • Bleeding
  • Infection
  • Adverse reaction to anaesthesia
  • Increased chance of hernia developing at the incision sites

Renal Health

Guide to Kidney Stone Disease & Diet

At the helm of non-pharmacological preventative therapies for kidney stones; also known as renal calculi, nephrolithiasis, or urolothiasis (, Kidney stones – Symptoms and causes, 2021), are dietary and lifestyle changes, as well as vitamin supplementation. 

Kidney stones can be defined as hardened masses that consist of crystallised minerals and salts, generated by concentrated urine. Stones too large to pass through the urinary system require medical treatment or intervention. Kidney stone disease is a common finding in today’s society, Studies show that, “It is estimated that one in ten people will have a kidney stone at some point in their lives.”(, Kidney Stones, 2021)

Kidney stones form due to various factors such as; obesity, genetics, lifestyle and certain medical conditions. A multitude of studies and trials have proven the link between the increased/decreased consumption or the exclusion of certain minerals/nutrients, and the reduced risk of kidney stone formation.

This article is based on the publication by, Ita P. Heilberg and David S. Goldfarb, titled Optimum Nutrition for Kidney Stone Disease. The following summarises their writings on the impact of certain dietary intake (Calcium, Oxalate, Protein, Sodium, Citrate & Potassium, Beverages, Phytate, and Calories & Fructose) on the risk of kidney-stone formation. Sharing their notes on the impact of the aforementioned factors on specific stone types, being; Calcium Oxalate Stones, Calcium Phosphate Stones, Uric Acid Stones, Cystine Stones, and Struvite Stones.

Photo by Ella Olsson on



Calcium (Ca) is one of the most important minerals required by the human body to maintain optimal performance of functions such as:

  • Growth and maintenance of healthy, strong bones.
  • Neurotransmission – maintaining healthy communication throughout the body.
  • Muscle movement. 
  • Cardiovascular movement. 

According to Heilberg and Goldfarb, calcium over-saturation of the urine is one of the biggest risk factors for calcium nephrolithiasis (Heilberg and Goldfarb, 2013). Calcium based stones account for up to 65-70% of kidney stones.

High levels of calcium present in the urine is referred to as hypercalciuria, and occurs in secondary and idiopathic incidences.

  • Secondary: Resulting as a side effect of another condition that causes excessive levels of calcium in the bloodstream.
  • Idiopathic: Occurring on its own, with normal levels of calcium present in the bloodstream. A complex primary metabolic alteration. At least half of calcium based stone formers are found to have idiopathic hypercalciuria.
  • An increase in active calcium transport by the intestines is observed in individuals with idiopathic hypercalciuria (IH). Seeing as; regardless of calcium intake, absorption of calcim is higher in IH patients. 
  • Commonly observed in IH patients:
    • A decrease in bone mineral density
    • High bone reabsorption
    • Reduced bone formation
  • Patients with IH excrete more calcium than was previously ingested when confronted with low calcium intake. The source of the excess calcium is most likely derived from bone.
  • Therefore, the entirety of effects decreasing calcium intake in IH stone forming patients has not yet been established.
  • Adversely to IH stone formers; decreasing dietary calcium intake in all other incident stone formers, reduces the risk of stone formation.
  • However, it has been widely observed that low calcium intake in young men, and younger and older women; can result in a 34% higher risk of developing kidney stones.
Sources of Calcium

  • Milk, cheese, cream and other dairy products.
  • Certain vareties of beans and lentilsLeafy vegetables eg. Spinach or kale
  • Fortified floursSeafoodSeeds eg. Sesame, poppy, chia and celery
  • Soya products, particularly those with added calcium.
  • Tofu

  • Oxalate


    Oxalate is a naturally occurring byproduct of metabolic activity, and derives from dietary sources. Excess urinary oxalate has the potential to collect in the kidneys and combine with other minerals present – leading to stone formation.

    • Urinary oxalate absorption is also dependant on calcium intake.
    • The malabsorption of fat is also responsible for increasing intestinal absorption of oxalate in conditions whereby reducing dietary fat might be considered.
    • It has been observed that in comparison to reducing oxalate intake, a more effective way of reducing urinary oxalate excretion is to increase calcium intake, especially if initial regular calcium intake is low.
    • The colonisation of Oxalobacter formigenes is another variable to factor into the consideration of the importance of dietary oxalate. Oxalobacter formigenes are obligate oxalate-degrading anaerobes [bacterium], found within the normal microbiome.
      • It’s presence in the colon is linked to a lower urinary oxalate excretion.
    • The significance of dietary oxalate restriction is yet to be determined in the overall prevention of stone formation. The exception being conditions such as bariatric surgery.
    • However, probiotic ingestion in patients characterised by high oxalate absorption resulted in the highest expectancy to experience clinically significant reductions in urinary oxalate. 
    • Which suggests that dietary oxalate plays a key role as a determinant of urinary oxalate excretion in response to the use of probiotic.
    Sources of Oxalate
    • Beans
    • Beer
    • Beets
    • Berries
    • Black tea
    • Chocolate
    • Cocoa powder
    • Coffee
    • Fizzy drinks/Soda
    • Green leafy vegetables
    • Nuts eg. Almonds and cashews
    • Okra
    • Oranges
    • Rhubarb
    • Soy beans & milk
    • Stevia sweetener
    • Sweet potato
    • Tofu
    • Wheat bran



    Protein is a fundamentally essential nutrient the body requires. Two of the most important roles played by protein are; providing the body with a source of fuel; and facilitating healing and growth, as protein is one of the main constituents for the formation of bodily tissue. However, sources of protein derived from animals has been proven to have a prevalent effect on many urinary variables that lead to stone formation.

    • The combination of a low-calcium diet with a high-animal-protein diet induces a negative calcium balance which is exceptionally harmful.
    • High animal-protein intake places one at risk of developing calcium based stones as it is a contributing factor to hyperuricosuria.
      • Hyperuricosuria – excessive amounts of uric acid present in the urine. 
      • Hyperuricosuria can in turn lead to hypocitraturia
      • Hypocitraturia – abnormally low urinary citrate excretion.
    • The induction of hypercalciuria by animal-protein intake occurs as a result of higher bone resorption (The removal of calcium from the bones) and lower tubular calcium reabsorption (A higher loss of calcium in the urine because the kidneys reclaim less calcium from the urine.). 
    • Although, it has been established that the acid load accompanying high animal-protein intake is not responsible for hypercalciuria.
    • In patients with nephrolithiasis, a short-term reduction of animal-protein intake, for up to approximately two weeks has yielded a significant increase in citrate excretion and a reduction of urinary excretion of calcium, phosphate, hydroxyproline, uric acid, and oxalate.
    • In spite of the significant role increased animal-protein intake plays in adverse fluctuations in urine chemistry; thus increasing the risk of kidney stones – the only diet restricting animal-protein intake to effectively reduce stone risk, entailed higher calcium intake and a reduction of sodium intake.
    Sources of Protein
    • Animal products eg. Beef, pork, poultry and fish/seafood
    • Animal byproducts eg. Dairy products and eggs etc.
    • Beans
    • Legumes
    • Nuts
    • Seeds
    • Soy



    In order for our bodies to function properly and optimally, we require a relatively low intake of sodium (Na). Sodium is both mineral and electrolyte, playing roles in:

    • The regulation of the body’s water and electrolyte balances
    • Muscle function
    • Nerve function
    • Electrical impulses throughout the body
    • Regulation of blood pressure and volume

    • A diet consisting of high sodium intake in patients with reduced proximal sodium reabsorption, will lead to decreased kidney tubular calcium reabsorption. (The kidney’s ability to remove calcium from the urine.)
    • Considerable changes to dietary sodium intake reflected direct variations in daily urinary calcium excretion.
    • Stone-formation have been observed to experience a deleterious effect on calcium and bone loss.
    • It’s been observed that a higher calcium, and reduced sodium intake is linked to the reduction of calciuria. 
      • Calciuria: calcium presence in the urine.
    Sources of Sodium
    • Biscuits
    • Cheese
    • Chicken
    • Cured meats
    • Foods containing vegetable preservatives and additives
    • Multi-ingredient meals eg. Pizza, filled sandwiches, macaroni and cheese, and frozen meals
    • Popcorn
    • Salad dressing
    • Processed/refined snacks eg. Crisps/chips
    • Sauces/condiments

    Citrate & Potassium


    Citrate: is a key component of metabolism. Derived from the three carboxy groups of citric acid (Pubchem, 2021). Essentially, citrate is the salt form of citric acid. Citric acid/citrate play important roles throughout the body; however, in context of kidney function and stone disease – urinary citrate inhibits the growth of calcium oxalate stones, and increases the solubility of calcium salts in the kidneys. 

    Potassium: is a naturally occurring mineral the human body requires. Potassium is also classified as an electrolyte; due to the reaction that takes place when introduced to water, which is the production of positively charged ions. Potassium weighs in as one of the top three most abundant minerals found in the human body. Playing roles in: 

    • Fluid regulation
    • Regulation of muscle contraction and function
    • Neurotransmission

    The health benefits of a potassium-rich diet have been proven to be significant; potentially reducing blood pressure and alleviating water retention; also aiding in the prevention of stroke, osteoperosis and kidney stones.

    • Multiple factors plays a role in the regulation of kidney citrate excretion; however, acid-base variables were found to have the biggest impact.
      • Acid-base: refers to the reaction that takes place between an acid (low pH) and a base (high pH), which results in a salt (neutral pH)
    • Acid-loads and acidosis aid in reducing ones risk of uric acid and cystine-based calculi. Both increase urinary citrate excretion by increasing kidney tubule reabsorption of citrate. Thus, reducing calciuria.
    • Stone formers with increased/high animal protein intake, are encouraged to substitute this intake with fruits and vegetables.
    • An alternative nonpharmacological therapy to potassium citrate is an increased intake of citrus fruits such as:
      • Oranges
      • Lemons
      • Limes
      • Certain tangerines
    • Being sources of natural dietary citrate, the intake of these citrus fruits may aid the management of hypocitraturia and/or uric acid and cystine stones.
    • Citrate found in both orange and grapefruit have been noted to increase urinary pH.
    • Drinking freshly squeezed citrus fruit juice delivers a similar amount of dietary citrate whilst also increasing fluid intake and output. However, it is not advised to cosnume processed, commercial fruit juice in high quantities due to their often high; caloric, fructose and/or carbohydrate content.
      • However, it has been observed that orange juice is an exception, as it has no significant effect on the risk of stone formation.
    • Increased fluid intake = increased fluid output
    • By increasing ones fluid intake and in turn output, urine supersaturation is reduced. Thereby, reducing the risk of kidney stones.
    • Another alternate non-pharmacological therapy for hypocitraturic stone formers is the increased intake of non-citrus alkaline fruits such as melon.
    • Various melons provide high loads of; potassium, malate and citrate. The consumption of which can result in an increase of citrate excretion.
    • In contrast, observational studies indicated that a higher potassium intake is linked to incident stones in males and older women.
    Sources of Citrate
    • Beans
    • Berries
    • Citrus fruits and juice eg. Lemons and limes contain the most citric acid, then oranges & grapefruit.
    • Melon
    Sources of Potassium
    • Animal products eg. Beef, fish and poultry
    • Certain fruit and vegetable juices
    • Fruits eg. Bananas, oranges and apricots
    • Legumes
    • Nuts
    • Vegetables eg. Cooked spinach/broccoli, mushrooms, zucchini, cucumber and peas

    Other Beverages

    • Increasing water intake to ensure a urinary output volume of approximately 2.5 litres per day, is associated with reduced risk of stone reccurance.
    • Although the previous point advocating the consumption of water in stone formers, is tried and true; certain variables may be reasons to be concerned about the, “effect of water hardness on kidney stone incidence.” Consuming inordinate/excessive amounts of water can have adverse effects on the kidneys.
      • Water “hardness”, merely refers to the mineral content of the water. Noteably calcium and magnesium. The “harder” the water, the higher this mineral content is.
    • The ingestion of mineral waters that deliver loads of bicarbonate and magnesium may reduce ones risk of of uric acid percipitation as the minerals may result in;
      • Favourable changes in urine pH.
      • Favourable changes in magnesium and citrate excretion.
      • Favourable changes to inhibitors of calcium oxalate stone formation.
      • Favourable changes to counterbalancing increased calcium excretion.
    • Caffeinated as well as decaffeinated tea and coffee have been observed to reduce the risk of stone formation.
    • (In moderation) Alcohol (specifically beer) has been associated with the reduced risk of kidney stone formation. Possibly as a result of decreased urinary concentration due to alcohols ability to inhibit the secretion of antidiuretic hormone (ADH). Leading to an increased need to urinate.



    Phytate or insitol hexaphosphate, inhibits calcium salt crystallisation and stone growth (specifically in vitro).

    • Many phytate rich foods (Such as; beans, cereal, whole grains and rice) are rich in oxalate content too.
    • It is possible that phytate alleviates/nullifies oxalate content – “induced lithogenic potential.”
    • Although only roughly 5% of ingested phytate appears in urinary output (implying that phytate is mostly nonabsorpable), the increased consumption of dietary phytate is strongly associated with increases in urinary excretion. Thus decreasing ones risk of kidney stone formation.
    Sources of Phytate
    • Beans
    • Grains
    • Legumes
    • Nuts
    • Seeds

    Calories & Fructose

    The caloric content (amount of calories) of a food or meal refers to the amount of energy the food delivers to the body. A calories is characterised by its ability to raise the temperature 1 gram of water by 1° Celsius, and is defined by the amount of energy this requires. Regular high calorie intake, exceeding ones needs will result in weight gain, as the body stores excess calories as fat. Fat is a readily available source of fuel and energy reserved in the body, one of the reasons why a certain percentage of body fat; appropriate to ones heigh, weight and age, is integral in the body’s ability to function optimally. A lower caloric intake can result in weight loss as the body will burn fat in order to replace the fuel it’s not getting from dietary caloric intake. 

    Fructose is a simple sugar that the liver converts into glucose, which is another main source of fuel for the body. Fructose makes up 50% of table sugar, which is why high fructose intake can result in abnormal metabolic activity.Vegetables and sweet fruits (and fruit in general) contain relatively low amounts of dietary fructose.

    • Many observational and epidemiological studies link, obesity, weight gain, insulin resistance, metabolic syndrome and diabetes with an increased prevalence of kidney stones.
    • A higher body mass index (BMI) is associated with:
      • Lower urine pH and increased risk of uric acid stones.
      • Increased excretion of urinary oxalate.
    • Ergo, obesity (one of the causes for a high BMI) has been linked to calcium oxalate stone formation.
    • Uric acid stone formers have been known to have an increased net-acid excretion and a decreased urine pH, compared to non-uric acid stone formers at any given level of urine sulphate excretion. 
    • This in turn means that another nonpharmacological treatment known as a process called alkalinisation (the neutralisation of an acid) has been observed to fail in uric acid stone formers.
    • Weight loss has been associated with reduced risk of formation of all compositions of stone.
    • Stone formers are recommended to avoid diets that combine intakes of high animal-protein with low- carbohydrate, such as the Atkins diet. Due to the following:
      • Reduced urine pH
      • Reduced citrate excretion
      • Increased uric acid excretion
    • Instead, stone formers are encouraged to follow a DASH diet, which stands for Dietary Approaches to Stop Hypertension. A low calorie DASH-style diet is rich in fruits and vegetables.
    • Although the consumption of fructose is not definitively linked to kidney stone formation, it has been observed that high fructose intake is associated with an increased risk of incident kidney stone formation.

    The Take Away

    The following summary is directly quoted from the source document; Optimum Nutrition for Kidney Stone Disease, written by Ita P. Heilberg and David S. Goldfarb

    Calcium Oxalate Stones

    “Idiopathic oxalate stone-formers are advised to reduce animal protein, oxalate and sodium in their diets as well as maintain adequate intake of calcium and increasing their consumption of citrate and potassium.”

    Calcium Phosphate Stones

    “Reduce their sodium intake to reduce calcium excretion.”

    Uric Acid Stones

    “The mainstay of therapy is weight loss and urinary alkalinisation, provided by a more vegetarian diet, leading to an increase in urine citrate content and pH. Reduction in animal protein intake may further reduce purine ingestion and uric acid excretion.”

    Cystine Stones

    “Restrict animal protein to reduce cystine methionine ingestion, and restrict sodium intake to further reduce excretion and supersaturation of cystine. Ingestion of vegetables high in content of organic anions, such as citrate, should be associated with higher urine pH.”

    Struvite Stones

    “Because of their infectious origin, diet has no definitive role for Struvite stones.”